Lowes timing7/8/2023 ![]() We observed a relative absence of association between the IQ scores of children with ASD and those of their first-degree relatives (parents: P =. We note that the mean IQ scores of the children who were testable (78.9 ± 21.7) were commensurate with (and somewhat higher than) their mean standardized scores for adaptive functioning on the Vineland-II (68.4 ± 11.5). 06Ĭhildren with ASD and their first-degree relatives exhibited a very broad IQ distribution (see Supplemental Figs 2 and 3, Supplemental Tables 7 and 8). Scheduling conflicts between parent or caregiver and professionalsĭifficulties due to lack of transportation to appointments Lack of available places or professionals to receive an evaluation in their area ![]() Parental level of education: college degreeĮxperienced a significant wait time to see a professionalĬosts associated with the evaluation and diagnostic process Median household income at time of first concerns Verbal Versus Nonverbal Subjects t tests dĪge in months at study enrollment, mean (SD)Īge in months at parental first concerns, mean (SD)Īge in months parent first shared concerns with a professional, mean (SD)Īge in months at initiation of services, mean (SD)Īge in months child received ASD diagnosis mean (SD) ![]() Note that for all sites, the average age of initiation of service use was 1 to 3 years earlier than ASD diagnosis, suggesting that the services delivered were unlikely to have been specific for autism. Subjects with comorbid ID tended to be diagnosed earlier than those without ID ( P <. Age at diagnosis ranged from an average of 54 months at the New York site to 80 months at the St Louis site. There was considerable variation across the recruitment sites, with diagnostic delays ranging from an average of 32 months (Los Angeles) to 53 months (St Louis), and proportion of subjects with ID ranging from 12.5% (New York) to 49.1% (Atlanta, where median household income and level of parental education of enrolled subjects was highest). In this sample, 98.2% of the families reported having some type of insurance coverage at the time of first concerns (49.3% private, 45.8% public, and 4.7% other). The mean age of diagnosis (64.9 months) came, on average, >3 years after parents reported having initial concerns about their child’s language, behavior, or development (mean = 23.0 months ± 17.9). We summarize in Table 2 the nature of delays in diagnosis as a function of key demographic characteristics, segregated by level of functioning of the subjects. Fourth, because of the nature of the parent study, we were able to specify the genetic ancestry of the study population. Third, as a direct validation of the CDC epidemiological surveillance statistics, and a check on the population-representativeness of the sample of AA children consecutively enrolled in this data collection, we seperately analyzed the subset of children who fell within the birth years and Missouri catchment area for the CDC surveillance program, incorporating data from the later. 10 The second involved direct cognitive assessment of the children and their close relatives this allowed a direct test of association of traditional correlates of low IQ with the cognitive outcomes of the children with ASD in this study, who manifested a wide and fully representative range of IQ for children with ASD. The first was an event history calendar interview on the pathway from earliest recognition of developmental delay to each child’s diagnosis. ![]() With the analyses presented in this report, we incorporated 4 elements of data acquisition.
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